|Posted on Tuesday, March 13, 2012 - 8:24 am: || |
Can anyone access the actual study? I'm curious about two things:
Were the glutamate receptors destroyed by excitotoxic reactions?
Were glutamate blockers used to prevent receptor loss?
|Posted on Tuesday, March 13, 2012 - 12:39 pm: || |
Jennifer, I think you're looking for this:
|Posted on Tuesday, March 13, 2012 - 8:02 pm: || |
Thank you, Roy.
Quickly scanning it, they used RU486 to prevent receptor loss. A powerful glutamate blocker. That doesn't prove my first question, but I sure suspect it's so. Will read it in depth later.
|Posted on Saturday, March 17, 2012 - 8:54 am: || |
Hmmm...I was thinking of MK801. In the article it stated : "Systemic injections of the GR antagonist RU486 blocked the
decreasing effect of repeated restraint stress on AMPAR-EPSC(Figure 2G,....."
So I wonder if RU486 has other properties besides the commonly known use...
|Posted on Sunday, March 18, 2012 - 12:59 am: || |
The most interesting theory about this biochemistry has been developed by professor Martin Pall. He offers a comprehensive model for the dysfunctional molecular pathways for a range of problems, including MSG sensitivity.